.css-1xsl8rf{width:100%;display:-webkit-box;display:-webkit-flex;display:-ms-flexbox;display:flex;-webkit-align-items:center;-webkit-box-align:center;-ms-flex-align:center;align-items:center;position:relative;overflow:hidden;background:var(--alert-bg);-webkit-padding-start:var(--chakra-space-4);padding-inline-start:var(--chakra-space-4);-webkit-padding-end:var(--chakra-space-4);padding-inline-end:var(--chakra-space-4);padding-top:var(--chakra-space-1);padding-bottom:var(--chakra-space-1);--alert-fg:var(--chakra-colors-orange-600);--alert-bg:var(--chakra-colors-orange-100);font-weight:var(--chakra-fontWeights-semibold);padding-left:var(--chakra-space-4);}.chakra-ui-dark .css-1xsl8rf:not([data-theme]),[data-theme=dark] .css-1xsl8rf:not([data-theme]),.css-1xsl8rf[data-theme=dark]{--alert-fg:var(--chakra-colors-orange-200);--alert-bg:rgba(251, 211, 141, 0.16);}@media screen and (min-width: 48em){.css-1xsl8rf{padding-left:var(--chakra-space-8);}}Please enable Javascript to use all features and improve your user experience.

Professor Mirko Trilling

Essen / DE

Universitätsklinikum Essen der Universität Duisburg-Essen

Speaker

25/03/2024
16:45 – 17:00 12 Min. 3 Min.
Abstract talk
O 011
The human cytomegalovirus-encoded pUS28 antagonizes CD4+ T-cell recognition by targeting CIITA
Adaptive immunity

Further involvements

25/03/2024
Poster
P 445
A spike mutation compatible with SARS-CoV-2 replication in natura confers resistance to fusion peptide-binding antibodies
Respiratory viruses
25/03/2024
16:45 – 17:00 12 Min. 3 Min.
Abstract talk
O 011
The human cytomegalovirus-encoded pUS28 antagonizes CD4+ T-cell recognition by targeting CIITA
Adaptive immunity
25/03/2024
18:15 – 18:30 12 Min. 3 Min.
Abstract talk
O 033
People living with HIV exhibit an exceptionally high prevalence and effector strength of cytomegalovirus-specific antibody responses
Virus infections and the immunocompromised host
27/03/2024
09:45 – 10:00 12 Min. 3 Min.
Abstract talk
O 063
Human cytomegalovirus entry and virus-specific CD4+ T cells induce cytokines that synergistically impair acute myeloid leukemia cell proliferation by interferon-dependent proteome alterations
Innate immunity